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Full article: The acetaminophen metabolite N-acetyl-p-benzoquinone imine ( NAPQI) inhibits glutathione synthetase in vitro; a clue to the mechanism of 5-oxoprolinuric acidosis?
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The metabolic pathways of acetaminophen (APAP) in the human body. The
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Fasting depletes Glutathione, turning "safe" Paracetamol doses into a liver-toxic emergency. Don't let a therapeutic dose become a lethal one. See the safety protocols on BMJ Best Practice via bio. 🔍 #MedEd #
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Clinical toxicity of Acetaminophen
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Q. The LD 50 of acetaminophen is increased in mice with deletions of genes for: 1 . Glutathione synthetase . 2 . CYP 1 A2 and CYP 2 E 1 . 3 . Glucuronyl transferase. 4 . N -acetyl transferase. 5 . Ah receptor
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NAPQI (N-Acetyl-4-benzoquinone Imine) | Acetaminophen Metabolite
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SUPER EASY MEMORY Too much Tylenol → More NAPQI → Less glutathione → Liver dies NAC → More glutathione → Liver saved
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